Home
About
Overview
Sharing Data
ORCID
Help
History (5)
An assessment of Health Care Information and Management Systems Society and Leapfrog data on computerized provider order entry.
Biochemical features of a patient with Zellweger-like syndrome with normal PTS-1 and PTS-2 peroxisomal protein import systems: a new peroxisomal disease.
Cardiopulmonary mechanoreceptors and renin release in humans.
Discovery of LYC-55716: A Potent, Selective, and Orally Bioavailable Retinoic Acid Receptor-Related Orphan Receptor-? (ROR?) Agonist for Use in Treating Cancer.
Proteomic Analysis of Plasma from California Sea Lions (Zalophus californianus) Reveals Apolipoprotein E as a Candidate Biomarker of Chronic Domoic Acid Toxicosis.
See All 5 Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
Search Result Details
Back to Search Results
This page shows the details of why an item matched the keywords from your search.
Search Results
Judge, Daniel
One or more keywords matched the following items that are connected to
Judge, Daniel
Item Type
Name
Concept
Myocytes, Cardiac
Academic Article
Multilevel analyses of SCN5A mutations in arrhythmogenic right ventricular dysplasia/cardiomyopathy suggest non-canonical mechanisms for disease pathogenesis.
Academic Article
Neonatal Transplantation Confers Maturation of PSC-Derived Cardiomyocytes Conducive to Modeling Cardiomyopathy.
Academic Article
Missense mutations in plakophilin-2 cause sodium current deficit and associate with a Brugada syndrome phenotype.
Academic Article
Mutations in Alstr?m protein impair terminal differentiation of cardiomyocytes.
Academic Article
Therapeutic Modulation of the Immune Response in Arrhythmogenic Cardiomyopathy.
Academic Article
Tnni3k alleles influence ventricular mononuclear diploid cardiomyocyte frequency.
Academic Article
Exercise triggers CAPN1-mediated AIF truncation, inducing myocyte cell death in arrhythmogenic cardiomyopathy.
Academic Article
The prevalent I686T human variant and loss-of-function mutations in the cardiomyocyte-specific kinase gene TNNI3K cause adverse contractility and concentric remodeling in mice.
Search Criteria
Myocytes Cardiac